Environmental Health

Table 5 Association between cardiovascular endpoints and PM _2.5 exposure in sensitivity analysis ^a,b

From: Secondhand tobacco smoke exposure and heart rate variability and inflammation among non-smoking construction workers: a repeated measures study

Variables	Post		Next morning
	β Coefficient (SE)	p Value	β Coefficient (SE)	p Value
^cHeart rate(bpm)	3.1(0.5)	0.239	−0.4(0.7)	0.680
^cSDNN(msec)	−8.1(3.0)	0.010	2.7(2.9)	0.361
^crMSSD(msec)	−8.2(3.9)	0.001	1.7(2.7)	0.538
^cLF(msec²)	−18.0(63)	<0.001	4.7(6.4)	0.471
^cHF(msec²)	−29.3(10.2)	0.006	3.7(6.1)	0.551
^cCRP(mg/L)	8.3(9.9)	0.464	25.7(9.3)	0.403
^cLymphocytes(k/μl)	1.7(2.2)	0.520	−4.5(2.8)	0.179
^dRed blood cells(M/μl)	−0.001(0.018)	0.946	−0.015(0.026)	0.575
^dWhite blood cells(k/μl)	0.433(0.146)	0.208	0.177(0.210)	0.426
^dNeutrophils(k/μl)	−0.041(0.108)	0.744	0.058(0.122)	0.661
^dMonocytes(k/μl)	−0.013(0.023)	0.630	−0.003(0.184)	0.145
^dBasophils(k/dl)	0.001(0.001)	0.506	−0.053(0.165)	0.762
^dEosinophils(k/μl)	0.018(0.006)	0.115	−0.004(0.012)	0.740
^dPlatelets(k/μl)	−0.639(2.853)	0.830	−3.07(2.65)	0.291

^aLinear mixed effects regression models were used to investigate the exposure-related changes in HRV(N = 94), CRP(N = 47) and whole blood cell count(N = 49). All models were adjusted for BMI, age, and circadian variation.
^bTwo participants who reported taking statin or beta-blockers were excluded in sensitivity analysis.
^cVariables were log-transformed due to the skewed distribution of model residuals. Associations were calculated by the equation percentage(%) = [exp(beta coefficients)-1]*100 and are interpreted as percent change.
^dVariables are untransformed and beta coefficients are presented from mixed regression models.

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ISSN: 1476-069X

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