Environmental Health

Table 4 Association between cardiovascular endpoints and PM _2.5 exposure ^a

From: Secondhand tobacco smoke exposure and heart rate variability and inflammation among non-smoking construction workers: a repeated measures study

Variables	Post		Next morning
	β Coefficient (SE)	p Value	β Coefficient (SE)	p Value
^bHeart rate(bpm)	2.9(0.5)	0.241	−0.3(0.7)	0.689
^bSDNN(msec)	−7.8(3.0)	0.012	2.5(2.9)	0.389
^brMSSD(msec)	−8.0(3.9)	0.048	1.7(2.6)	0.516
^bLF(msec²)	−17.2(6.3)	0.010	4.9(6.3)	0.441
^bHF(msec²)	−29.0(10.1)	0.006	3.8(6.0)	0.525
^bCRP(mg/L)	8.1(9.3)	0.445	24.2(9.0)	0.043
^bLymphocytes(k/μl)	1.7(2.1)	0.501	−3.1(2.6)	0.353
^cRed blood cells(M/μl)	−0.004(0.017)	0.852	−0.011(0.025)	0.673
^cWhite blood cells(k/μl)	0.424(0.144)	0.021	0.200(0.203)	0.356
^cNeutrophils(k/μl)	−0.038(0.11)	0.756	0.076(0.118)	0.556
^cMonocytes(k/μl)	−0.012(0.022)	0.655	−0.027(0.018)	0.211
^cBasophils(k/dl)	0.001(0.001)	0.521	−0.003(0.016)	0.862
^cEosinophils(k/μl)	0.018(0.006)	0.106	−0.004(0.012)	0.737
^cPlatelets(k/μl)	−0.808(2.72)	0.778	−2.27(2.53)	0.404

^aLinear mixed effects regression models were used to investigate the exposure-related changes in HRV(N = 97), CRP(N = 50) and whole blood cell count(N = 52). All models were adjusted for BMI, age, and circadian variation.
^bVariables were log-transformed due to the skewed distribution of model residuals. Associations were calculated by the equation percentage (%) = [exp(beta coefficients)-1]*100 and are interpreted as percent change.
^cVariables are untransformed and beta coefficients are presented from mixed regression models.

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ISSN: 1476-069X

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