As Soleman SR et al. pointed out, the radiation dose is decreasing, but at the same time, it is certain that it remains in the long-term. Therefore, many Fukushima people are still forced to evacuate. Furthermore, radiation detriment is characterized by the fact that damage continues to occur for a long time after exposure. It is clear from the Life Span Study in Hiroshima and Nagasaki that not only carcinogenesis but also heart disease, respiratory disorders, and digestive disorders continue to occur for a long period of time . Similarly, numerous studies report reproductive detriment concerning, e.g., stillbirths, perinatal deaths, birth defects, and chromosome aberrations demonstrating that radiation injury persisted for many years [5,6,7,8,9,10,11,12,13,14,15,16,17]. The elevation of LBW over long periods, despite declining radiation doses, is a hallmark of radiation injury.
In their second criticism, Soleman et al. overlooked that our estimated increase of the LBW proportion after 2011 is only the average of an effect without respect to and quantification of any temporal pattern of the LBW increase after Fukushima. To illustrate this, we analyzed the scenario of our Fig. 4D  concerning Fukushima, Miyagi, Ibaraki, Tochigi, and Iwate over a symmetrical period (2004 to 2018) allowing
for a simple jump from 2012 onward, and
for the interaction of this jump with time (technically speaking).
For according point estimates, interval estimates, and p-values of these analyses see Fig. 1 of this letter. It shows that the increase in LBWp decreases with time, but this decrease is not significant (p-value 0.1075) due to insufficient statistical power provided by the scenario of Fig. 4D . Therefore, this decrease of the increase in the LBWp in the five highly contaminated prefectures after Fukushima is also compatible with a constant effect, at least over the 7-year period 2012 to 2018.